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anti dusp1  (Santa Cruz Biotechnology)


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    Structured Review

    Santa Cruz Biotechnology anti dusp1
    Anti Dusp1, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 94/100, based on 518 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mkp-1+antibody/MKP-1+Antibody/pmc12595205-61-24-29
    Average 94 stars, based on 518 article reviews
    anti dusp1 - by Bioz Stars, 2026-09
    94/100 stars

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    other:

    Article Title: Fructose Protects Murine Hepatocytes from Tumor Necrosis Factor-induced Apoptosis by Modulating JNK Signaling
    Article Snippet: Agonistic CD95 antibody (clone Jo-2) was purchased from BD Pharmingen (San Diego, CA), Bid antibody, p-JNK, and p-CREB were from Cell Signaling (Boston, MA), HRP-coupled rabbit anti-goat was from Dako (Glostrup, Denmark), c-FLIP l was from Enzo (New York, NY), HRP-coupled goat anti-rabbit was from Dianova (Hamburg, Germany), agarose-coupled JNK1/JNK2, total JNK, p-c-jun, c-jun, and MKP-1 were from Santa Cruz Biotechnology (Santa Cruz, CA), murine TNF was from Innogenetics (Ghent, Belgium), KT 5720 was from Alexis (Lausen, CH), Bt 2 cAMP and ZM241385 were from Tocris Cookson (Bristol, UK), and 8-Br-cAMP-AM was from Biolog Life Science (Bremen, Germany).

    Article Title: HIV Impairs TNF-α Release in Response to Toll-Like Receptor 4 Stimulation in Human MacrophagesIn Vitro
    Article Snippet: The molecular mechanisms for increased risk of bacterial pneumonia in HIV persons remain incompletely understood.. Recognizing the critical role of Toll-like receptor (TLR) signaling in host defense, this study showed that human U937 macrophage stimulation by the TLR4-specific ligand, lipid A (biologically active component of bacterial LPS), promoted TNFrelease through extracellular regulated kinase (ERK)1/2 mitogen-activated protein (MAP) kinase phosphorylation.. In contrast, HIV U1 macrophages had significantly reduced TNFrelease (despite preserved TLR4 expression) and reduced ERK1/2 phosphorylation, whereas TNFrelease was intact via a TLR4-independent pathway.

    Article Title: Down-regulation of vitamin D receptor in mammospheres: implications for vitamin D resistance in breast cancer and potential for combination therapy.
    Article Snippet: MKP-1 and pERK1/2 antibodies were obtained from Santa Cruz Biotech (Santa Cruz, CA) and Cell Signaling (Danvers, MA) respectively.

    Article Title: T-kininogen, a cystatin-like molecule, inhibits ERK-dependent lymphocyte proliferation.
    Article Snippet: Plasma levels of kininogens increase with age in both rats and humans.. Kininogens are inhibitors of cysteine proteinases, and filarial cysteine proteinase inhibitors (cystatins) reduce the proliferation of T cells.. We evaluated whether T-kininogen (T-KG) might mimic this effect, and here we present data indicating that exposure of either rat splenocytes or Jurkat cells to purified T-KG results in inhibition of both ERK activation and [H]-thymidine incorporation, both basal and in response to ConA or PHA.

    Article Title: Mitogen-Activated Protein Kinase Kinase 4/c-Jun NH 2 -Terminal Kinase Kinase 1 Protein Expression Is Subject to Translational Regulation in Prostate Cancer Cell Lines
    Article Snippet: The antibodies and dilutions were as follows: MKK4 (1:5,000; Santa Cruz Biotechnology), c-myc (1:500; Cell Signaling), MKP-1 (1:500; Santa Cruz Biotechnology), tubulin (1:1,000; Santa Cruz Biotechnology), lamin A/C (1:2,000; Upstate), β-actin (1:10,000; Calbiochem), anti-rabbit horseradish peroxidase (1:10,000; Cell Signaling), and anti-mouse IgG-horseradish peroxidase (1:10,000; Sigma).

    Article Title: Nitric oxide regulates cytokine induction in the diaphragm in response to inspiratory resistive breathing.
    Article Snippet: Ioanna Sigala, Panayiotis Zacharatos, Stavroula Boulia, Dimitris Toumpanakis, Tatiana Michailidou, Dimitris Parthenis, Charis Roussos, Andreas Papapetropoulos, Sabah N. Hussain, and Theodoros Vassilakopoulos Department of Critical Care and Pulmonary Services, University of Athens Medical School, Evangelismos Hospital, Athens, Greece; G. P. Livanos and M. Simou Laboratories, Athens, Greece; Laboratory of Molecular Pharmacology, Department of Pharmacy, University of Patras, Patra, Greece; and Critical Care Division, Department of Medicine, McGill University Health Centre, McGill University, Montreal, Quebec, Canada

    Article Title: MKP-1 is a target of miR-210 and mediate the negative regulation of miR-210 inhibitor on hypoxic hPASMC proliferation.
    Article Snippet: Chonic hypoxia, smooth muscle cell (SMC) proliferation and vascular remodeling are hallmark features of pathogenic pulmonary artery hypertension.. MicroRNAs (miRNAs), endogenously expressed small noncoding RNAs, regulate gene expression at the post-transcriptional level.. MiR-210 is considered a “master miRNA” in the control of diverse functions in hypoxic cells and tissues and has a cytoprotective function in pulmonary artery SMCs during hypoxic stress.



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